Supraventricular tachycardia other name
Supraventricular tachycardia other name Supraventricular tachycardia, commonly abbreviated as SVT, is a rapid heart rhythm originating above the heart’s ventricles, specifically within the atria or the atrioventricular (AV) node. This condition causes the heart to beat faster than normal, often leading to symptoms such as palpitations, chest discomfort, lightheadedness, or even fainting. While SVT is generally not life-threatening, it can cause significant discomfort and, in some cases, may lead to more serious complications if left untreated.
The term “supraventricular tachycardia” is quite descriptive, with “supraventricular” indicating that the abnormal electrical activity begins above the ventricles, and “tachycardia” referring to an abnormally fast heart rate—typically over 100 beats per minute, with episodes often surpassing 150 bpm. Given its descriptive nature, SVT is sometimes also called “paroxysmal supraventricular tachycardia” or PSVT, especially when episodes occur suddenly and resolve spontaneously or with intervention. The word “paroxysmal” emphasizes the episodic or sudden nature of the arrhythmia.
In everyday language, people might refer to SVT simply as a “heart rhythm problem” or “heart flutters,” but medical professionals often use the terms SVT or PSVT to specify the condition precisely. Another less common term occasionally encountered is “atrioventricular nodal reentrant tachycardia” (AVNRT), which is a specific type of SVT caused by a reentrant circuit within or near the AV node. While AVNRT is a common cause of SVT, the broader term encompasses other mechanisms such as atrioventricular reciprocating tachycardia (AVRT) and ectopic atrial tachycardia. Supraventricular tachycardia other name
Supraventricular tachycardia other name The causes of SVT can vary. It may occur in healthy individuals without underlying heart disease, often triggered by stress, caffeine, alcohol, or fatigue. Conversely, underlying conditions such as heart disease, hyperthyroidism, or electrolyte imbalances can predispose individuals to episodes. Structural abnormalities, accessory pathways, or abnormal electrical circuits in the heart can also contribute to the development of SVT.
Diagnosis involves a detailed history, physical examination, and electrocardiogram (ECG). During an episode, the ECG typically shows a narrow-complex tachycardia with rapid, regular beats. Sometimes, the episodes can be terminated with vagal maneuvers—such as bearing down or holding one’s breath—and confirmed with electrical or pharmacological interventions if needed. Supraventricular tachycardia other name
Managing SVT can include acute treatments like vagal maneuvers and medications such as adenosine, which can quickly restore normal heart rhythm. For recurrent episodes, doctors might recommend longer-term solutions, including medications like beta-blockers or calcium channel blockers. In some cases, catheter ablation—a minimally invasive procedure—is performed to destroy the abnormal electrical pathway and potentially cure the condition. Supraventricular tachycardia other name
Supraventricular tachycardia other name Understanding the various names and mechanisms of SVT helps patients and healthcare providers communicate effectively. Recognizing that “SVT” and “PSVT” often refer to the same broad category of arrhythmias can facilitate prompt diagnosis and treatment, ultimately improving patient outcomes.
